Involvements of TRP Channels and Oxidative Stress in Pain (Record no. 41491)

MARC details
000 -LEADER
fixed length control field 02611naaaa2200349uu 4500
001 - CONTROL NUMBER
control field https://directory.doabooks.org/handle/20.500.12854/50653
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20220219202206.0
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 978-2-88945-595-9
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9782889455959
024 7# - OTHER STANDARD IDENTIFIER
Standard number or code 10.3389/978-2-88945-595-9
Terms of availability doi
041 0# - LANGUAGE CODE
Language code of text/sound track or separate title English
042 ## - AUTHENTICATION CODE
Authentication code dc
100 1# - MAIN ENTRY--PERSONAL NAME
Personal name Mustafa Naziroglu
Relationship auth
245 10 - TITLE STATEMENT
Title Involvements of TRP Channels and Oxidative Stress in Pain
260 ## - PUBLICATION, DISTRIBUTION, ETC.
Name of publisher, distributor, etc. Frontiers Media SA
Date of publication, distribution, etc. 2018
300 ## - PHYSICAL DESCRIPTION
Extent 1 electronic resource (126 p.)
506 0# - RESTRICTIONS ON ACCESS NOTE
Terms governing access Open Access
Source of term star
Standardized terminology for access restriction Unrestricted online access
520 ## - SUMMARY, ETC.
Summary, etc. Undoubtedly, pain conditions the quality of life of millions of people worldwide suffering a wide range of diseases. Major research efforts are being made by the international scientific community to determine the mechanisms underlying nociception. Growing evidence points out a complex network including oxidative and nitrosative stress, inflammatory response and cation signaling. In this sense, transient receptor potential (TRP) channels have attracted researchers’ attention. Expression levels are very different in tissues and cells mediating a myriad of processes in our organism. At the neurological level, it has been observed that the expression levels of four TRP channels (TRPA1, TRPM2, TRPV1, and TRPV4) are high in neurons related to nociception, including dorsal root ganglion and trigeminal ganglia neurons. For this reason, this research field promises to shed light on this intricated matrix linking oxidative stress, calcium signaling (via TRP channels), and inflammatory signals in different pain modalities, including neuropathic pain and chemotherapy-induced peripheral pain. In such a way, all this intense research activity will enable us to design individual and rational treatment strategies for pain relief, such as the use of molecular neurosurgery.
540 ## - TERMS GOVERNING USE AND REPRODUCTION NOTE
Terms governing use and reproduction Creative Commons
Use and reproduction rights https://creativecommons.org/licenses/by/4.0/
Source of term cc
-- https://creativecommons.org/licenses/by/4.0/
546 ## - LANGUAGE NOTE
Language note English
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term cation signaling
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term pain
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term nervous system
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term nociception
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term TRP channels
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term oxidative stress
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Jose A. Pariente
Relationship auth
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Laszlo Pecze
Relationship auth
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Cristina Carrasco
Relationship auth
856 40 - ELECTRONIC LOCATION AND ACCESS
Host name www.oapen.org
Uniform Resource Identifier <a href="https://www.frontiersin.org/research-topics/5551/involvements-of-trp-channels-and-oxidative-stress-in-pain">https://www.frontiersin.org/research-topics/5551/involvements-of-trp-channels-and-oxidative-stress-in-pain</a>
Access status 0
Public note DOAB: download the publication
856 40 - ELECTRONIC LOCATION AND ACCESS
Host name www.oapen.org
Uniform Resource Identifier <a href="https://directory.doabooks.org/handle/20.500.12854/50653">https://directory.doabooks.org/handle/20.500.12854/50653</a>
Access status 0
Public note DOAB: description of the publication

No items available.