Organogenesis From Development to Disease (Record no. 70455)

MARC details
000 -LEADER
fixed length control field 02448naaaa2200397uu 4500
001 - CONTROL NUMBER
control field https://directory.doabooks.org/handle/20.500.12854/55410
005 - DATE AND TIME OF LATEST TRANSACTION
control field 20220220062442.0
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 978-2-88945-318-4
020 ## - INTERNATIONAL STANDARD BOOK NUMBER
International Standard Book Number 9782889453184
024 7# - OTHER STANDARD IDENTIFIER
Standard number or code 10.3389/978-2-88945-318-4
Terms of availability doi
041 0# - LANGUAGE CODE
Language code of text/sound track or separate title English
042 ## - AUTHENTICATION CODE
Authentication code dc
100 1# - MAIN ENTRY--PERSONAL NAME
Personal name Misty Good
Relationship auth
245 10 - TITLE STATEMENT
Title Organogenesis From Development to Disease
260 ## - PUBLICATION, DISTRIBUTION, ETC.
Name of publisher, distributor, etc. Frontiers Media SA
Date of publication, distribution, etc. 2017
300 ## - PHYSICAL DESCRIPTION
Extent 1 electronic resource (110 p.)
506 0# - RESTRICTIONS ON ACCESS NOTE
Terms governing access Open Access
Source of term star
Standardized terminology for access restriction Unrestricted online access
520 ## - SUMMARY, ETC.
Summary, etc. During embryonic development there are many processes that must take place to produce a viable and healthy fetus. Alterations in the cellular and/or molecular interactions within any particular organ can cause catastrophic defects leading to defective organogenesis and death of the embryo. Even slight malformations of the organs can cause developmental reprogramming and increase the likelihood of adult onset diseases. The use of experimental animals with genetic mutations that mimic many human conditions has lead to significant scientific advancement of these complex and multifaceted diseases. Furthermore, the utilization of organ culture systems has made for profound insights into the formation of organs. Over recent years, the technology related to molecular profiling and imaging of developing organs has dramatically improved, leading to the identification of subtle genetic and phenotypic alterations. This research topic will focus on the area of organ formation and modeling of human developmental diseases.
540 ## - TERMS GOVERNING USE AND REPRODUCTION NOTE
Terms governing use and reproduction Creative Commons
Use and reproduction rights https://creativecommons.org/licenses/by/4.0/
Source of term cc
-- https://creativecommons.org/licenses/by/4.0/
546 ## - LANGUAGE NOTE
Language note English
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Disease Models
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term kidney development
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Bladder innervation
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Animal
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Bowel disease
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Inflammation
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Developmental Biology
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Diabetes Mellitus
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Organogenesis
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term Type 1
653 ## - INDEX TERM--UNCONTROLLED
Uncontrolled term bone mineralization
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Seppo Vainio
Relationship auth
700 1# - ADDED ENTRY--PERSONAL NAME
Personal name Sunder Sims-Lucas
Relationship auth
856 40 - ELECTRONIC LOCATION AND ACCESS
Host name www.oapen.org
Uniform Resource Identifier <a href="https://www.frontiersin.org/research-topics/3607/organogenesis-from-development-to-disease">https://www.frontiersin.org/research-topics/3607/organogenesis-from-development-to-disease</a>
Access status 0
Public note DOAB: download the publication
856 40 - ELECTRONIC LOCATION AND ACCESS
Host name www.oapen.org
Uniform Resource Identifier <a href="https://directory.doabooks.org/handle/20.500.12854/55410">https://directory.doabooks.org/handle/20.500.12854/55410</a>
Access status 0
Public note DOAB: description of the publication

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