TY  - GEN
AU  - Koch,Benjamin
AU  - Geiger,Helmut
AU  - Baer,Patrick C.
TI  - Kidney Inflammation, Injury and Regeneration
SN  - books978-3-03928-539-6
PY  - 2020///
PB  - MDPI - Multidisciplinary Digital Publishing Institute
KW  - microRNAs
KW  - n/a
KW  - transcription
KW  - ischemia/reperfusion injury
KW  - DSS-colitis
KW  - kidney inflammation
KW  - therapeutics targets
KW  - CXCL13
KW  - glomerulus
KW  - interleukin-6
KW  - rhabdomyolysis
KW  - IgA nephropathy
KW  - CREB Regulated Transcriptional Coactivators (CRTC)
KW  - slit diaphragm
KW  - injury
KW  - xanthine oxidase
KW  - Salt Inducible Kinase (SIK)
KW  - acute and chronic kidney disease
KW  - therapeutic target
KW  - KIT-IgA score
KW  - G-protein-coupled bile acid receptor (TGR5)
KW  - lysophosphatidic acid
KW  - glomerular injury
KW  - IL-18
KW  - mesenchymal stem cells
KW  - Taiwan
KW  - acute kidney injury
KW  - renal ischemia-reperfusion
KW  - long non-coding RNA
KW  - fibrosis
KW  - acute kidney failure
KW  - diabetic kidney diseases
KW  - chronic kidney disease
KW  - lncRNA
KW  - LPS-binding protein
KW  - endotoxemia-induced oliguric kidney injury
KW  - dapagliflozin
KW  - cPLA2 and COX-2
KW  - NLRP3 inflammasome
KW  - CmklR1
KW  - haem
KW  - chronic kidney injury
KW  - omega-3 fatty acid
KW  - noninvasive
KW  - inflammation
KW  - regulated necrosis
KW  - GLP-1 receptor agonists
KW  - miRNA
KW  - AKI
KW  - SGLT2 inhibitors
KW  - diabetic kidney disease
KW  - extracellular vesicles
KW  - podocin
KW  - type IV collagen
KW  - epithelial cells
KW  - nephrin
KW  - 2-kidney-1-clip
KW  - renal fibrosis
KW  - papilla
KW  - diagnostics
KW  - necrosis
KW  - non-coding RNAs
KW  - podocyte
KW  - Thy1.1 nephritis
KW  - KIT assay
KW  - oxidative stress
KW  - conditioned medium
KW  - C-reactive protein
KW  - pericyte
KW  - myofibroblast
KW  - Nuclear Factor kappa-light-chain-enhancer of activated B cells (NF-?B)
KW  - endotoxemia
KW  - modifier gene
KW  - polymorphism
KW  - renal stem cells
KW  - kidney
KW  - polyploidization
KW  - Class IIa Histone Deacetylases (HDAC)
KW  - 2k1c
KW  - molecular signaling
KW  - proximal tubule
KW  - arachidonic acid
KW  - empagliflozin
KW  - tubular injury
KW  - signaling cascade
KW  - signal transduction
KW  - inflammatory maker
KW  - niches
KW  - biomarkers
KW  - renal progenitors
KW  - type V collagen
KW  - cyclooxygenase
KW  - focal segmental glomerulosclerosis
KW  - inflammatory bowel disease (IBD)
KW  - chronic kidney disease (CKD)
KW  - allograft rejection
KW  - renovascular hypertension
KW  - genotype
KW  - molecular mechanisms
KW  - ROS
KW  - prediction
KW  - glomerular filtration barrier (GFB)
KW  - alport syndrome
KW  - scattered tubular cells
KW  - long non-coding RNAs
KW  - renal inflammation
KW  - lysophosphatidic acid receptor
KW  - cAMP Regulatory Element Binding Protein (CREB)
KW  - Farnesiferol B
KW  - differentiation
KW  - mesenchymal stromal cells
KW  - modified-MSCs
KW  - kidney transplantation
KW  - polyunsaturated fatty acids
KW  - apoptosis
KW  - type I collagen
KW  - diabetes mellitus
KW  - natural products
KW  - lipoxygenase
KW  - stem cell
KW  - T cell-mediated rejection
KW  - exosomes
KW  - renal injury
KW  - obese kidney fibrosis
KW  - kidney injury
KW  - cytotoxicity
KW  - mesenchymal stem cell
KW  - pigment nephropathy
KW  - mesodermal stem cell
KW  - ischemia-reperfusion
KW  - cytochrome P450
KW  - renal cell carcinoma
KW  - hematuria
KW  - B-cell attracting chemokine
KW  - microRNA
KW  - chemerin
KW  - glomerular basement membrane
KW  - glomerular damage
KW  - renal tubular cells
KW  - kidney proximal tubule
KW  - exosome
KW  - hypertension
KW  - diabetic nephropathy
N1  - Open Access
N2  - Acute kidney injury (AKI) is still associated with high morbidity and mortality incidence rates, and also bears an elevated risk of subsequent chronic kidney disease. Although the kidney has a remarkable capacity for regeneration after injury and may recover completely depending on the type of renal lesions, the options for clinical intervention are restricted to fluid management and extracorporeal kidney support. The development of novel therapies to prevent AKI, to improve renal regeneration capacity after AKI, and to preserve renal function is urgently needed. The Special Issue covers research articles that investigated the molecular mechanisms of inflammation and injury during different renal pathologies, renal regeneration, diagnostics using new biomarkers, and the effects of different stimuli like medication or bacterial components on isolated renal cells or in vivo models. The Special Issue contains important reviews that consider the current knowledge of cell death and regeneration, inflammation, and the molecular mechanisms of kidney diseases. In addition, the potential of cell-based therapy approaches that use mesenchymal stromal/stem cells or their derivates is summarized. This edition is complemented by reviews that deal with the current data situation on other specific topics like diabetes and diabetic nephropathy or new therapeutic targets
UR  - https://mdpi.com/books/pdfview/book/2165
UR  - https://directory.doabooks.org/handle/20.500.12854/51086
ER  -