| 000 | 01861naaaa2200265uu 4500 | ||
|---|---|---|---|
| 001 | https://directory.doabooks.org/handle/20.500.12854/62226 | ||
| 005 | 20220220082042.0 | ||
| 020 | _a9783038426554 | ||
| 020 | _a9783038426547 | ||
| 041 | 0 | _aEnglish | |
| 042 | _adc | ||
| 100 | 1 |
_aMarc Kvansakul (Ed.) _4auth |
|
| 245 | 1 | 0 | _aViral Infection and Apoptosis |
| 260 |
_bMDPI - Multidisciplinary Digital Publishing Institute _c2018 |
||
| 300 | _a1 electronic resource (270 p.) | ||
| 506 | 0 |
_aOpen Access _2star _fUnrestricted online access |
|
| 520 | _aApoptosis is a form of programmed cell death that enables the removal of damaged, infected, or otherwise unwanted cells in a controlled manner. Apoptosis can be initiated by multiple independent pathways that ultimately converge at a point where proteolytic enzymes belonging to the caspase family are activated, which dismantle the apoptotic cell. Multicellular organism have employed apoptotic mechanisms during host defence in response to viral infection to limit or prevent viral spread and replication. Consequently, viruses have evolved sophisticated molecular countermeasures to disarm host apoptotic defences, and this series of reviews and primary research articles in this Special Issue explores the intricate molecular interplay between viruses and their hosts when they battle for control of host apoptotic check-points. | ||
| 540 |
_aCreative Commons _fhttps://creativecommons.org/licenses/by-nc-nd/4.0/ _2cc _4https://creativecommons.org/licenses/by-nc-nd/4.0/ |
||
| 546 | _aEnglish | ||
| 653 | _ahost defence | ||
| 653 | _aApoptosis | ||
| 653 | _acaspase | ||
| 856 | 4 | 0 |
_awww.oapen.org _uhttp://www.mdpi.com/books/pdfview/book/503 _70 _zDOAB: download the publication |
| 856 | 4 | 0 |
_awww.oapen.org _uhttps://directory.doabooks.org/handle/20.500.12854/62226 _70 _zDOAB: description of the publication |
| 999 |
_c75636 _d75636 |
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